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Venous Sinus Stenosis and Pulsatile Tinnitus: Scans and Stenting

A narrowed vein inside the skull is a leading cause of pulsatile tinnitus. How it links to IIH, how scans and pressure tests find it, and what stenting does.

By Tinnitus Clarified TeamPublished 11 min read

Key takeaways

  • Narrowing of the transverse or sigmoid sinus is a leading venous cause of pulsatile tinnitus: a 2026 expert consensus calls it the most common cause, and it is closely tied to idiopathic intracranial hypertension.
  • In a 2003 MRI study, substantial narrowing on both sides appeared in 27 of 29 people with idiopathic intracranial hypertension but also in 4 of 59 controls, so a scan finding has to be matched to symptoms and pressures.
  • Catheter venography measures the pressure drop across the narrowing, and a gradient of about 8–10 mm Hg is the threshold most centres use before offering a stent.
  • In a 2026 meta-analysis of 850 patients, stenting resolved pulsatile tinnitus completely in 87.1%, with 10.9% recurrence. Major complications ran at 1.9% in a 474-patient review, and no randomised trial of stenting has yet reported.
  • A pulsing sound with a sudden severe headache, sudden neck pain, a drooping eyelid or loss of vision is an emergency. On its own, pulsatile tinnitus needs a doctor within a few weeks.

Get these checked without waiting

Most tinnitus is not an emergency. These signs are the exception: they need a prompt medical assessment rather than a wait-and-see.

  • A pulsing sound with a sudden severe headache or sudden neck pain
  • A pulsing sound with a drooping eyelid, loss of vision or new double vision
  • Sudden weakness or drooping in the face or an arm, or trouble speaking or seeing
  • Whooshing with headaches and vision that briefly greys out or blurs

Call your local emergency number for sudden weakness or drooping in the face, trouble speaking or seeing, or severe vertigo that will not settle.

When to See a Doctor checklist

Venous sinus stenosis is a narrowing of the transverse or sigmoid sinus, large veins inside the skull that drain the brain, and it is among the commonest findable causes of pulsatile tinnitus, often treatable with a stent.

The narrowing matters because of what it does to the blood passing through it. A 2026 cohort study in Neurosurgical Review describes venous sinus stenosis as producing turbulent venous flow and raised pressure in the veins, and the turbulence, close to the ear, is heard as a whoosh in time with the pulse. That is a real physical sound with a source, which is why pulsatile tinnitus is investigated rather than managed like the steady kind.

How a venous whoosh behaves

A 2026 review of cerebral venous disorders in Frontiers in Neurology describes venous pulsatile tinnitus as a whooshing in one or both ears that may change with head position, with straining, or when the jugular vein on the same side is pressed, the last being its key clue to a venous source. The review advises that the examination include listening for a bruit, a sound the clinician can hear too, which would make it objective tinnitus.

That does not say which venous problem is responsible. The review lists three: venous sinus stenosis, dural arteriovenous fistulas, and sigmoid sinus wall abnormalities, which can occur alongside a stenosis. High-grade fistulas can bleed into the brain, the review notes, which is why they are looked for specifically.

How common it is behind pulsatile tinnitus

A 2026 consensus statement from the ARISE II meeting, a roundtable of academic, government and industry experts, calls venous sinus stenosis the most common cause of pulsatile tinnitus. That is expert judgement rather than a measured prevalence, but controlled data point the same way.

A 2026 case-control study in AJNR compared 148 people with pulsatile tinnitus with 98 controls scanned for other reasons. Transverse sinus stenosis was significantly more common with pulsatile tinnitus (p < .01). Narrowing of the internal jugular vein lower in the neck was not: its prevalence did not differ between the groups.

In the 2026 Otology & Neurotology meta-analysis of stenting for pulsatile tinnitus, venous sinus stenosis accounted for 90.3% of the conditions treated.

Idiopathic intracranial hypertension (IIH) is raised pressure in the fluid around the brain with no identifiable cause. Its link with sinus narrowing is strong and was measured early.

In a 2003 study in Neurology, 29 people with established IIH and 59 controls had contrast-enhanced MR venography, graded blind by three readers. Substantial narrowing of both transverse sinuses appeared in 27 of the 29 people with IIH and in 4 of the 59 controls. The authors reported that the grading identified IIH with a sensitivity and specificity of 93%.

Which comes first is less settled. Catheter measurements published in 1995 found high pressure upstream in the sinuses of nine people with IIH, dropping sharply at the outer third of the transverse sinus. In 2002 the same team removed 20 to 25 mL of spinal fluid during the measurement, and the venous pressure fell back immediately. They concluded that raised pressure around the brain was squeezing the sinuses, not the reverse.

The ARISE II consensus says growing evidence connects stenosis with reduced reabsorption of spinal fluid and raised pressure in a substantial subset of people with IIH, so the two may feed each other. The Frontiers review separates intrinsic stenoses, linked to arachnoid granulations inside the sinus, from extrinsic ones caused by pressure from outside, which may respond differently to treatment.

For the tinnitus specifically:

  • In a 2024 meta-analysis of 694 people with IIH treated by stenting, 58.3% had tinnitus beforehand.
  • In a 2021 CT study, transverse sinus stenosis did not separate IIH patients with one-sided pulsatile tinnitus from those without it. Sigmoid sinus wall dehiscence and a dominant draining side did.
  • In a 2025 chart review of 132 people whose optic disc swelling from IIH had resolved, 97 (73%) had presented with pulsatile tinnitus, and 59 of those 97 (61%) still had it. Settling the eyes did not always settle the sound.
  • In a 2026 registry study, 33 of 517 people stented for IIH had normal pressure in the superior sagittal sinus, upstream of the narrowing, yet their tinnitus improved at a similar rate (78.3%, against 84.6% in matched patients with high pressure).

How it is found: MR venography, CT venography and catheter venography

The Frontiers review recommends going step by step:

  • MRI with MR venography is the preferred first test. It shows the venous sinuses, can show a fistula and signs of raised pressure, and involves no radiation.
  • CT venography and a fine-slice CT of the temporal bone add detail about the bone and the vein, which helps with sigmoid sinus wall abnormalities and with measuring how tight a narrowing is.
  • Catheter angiography remains the gold standard when a fistula is suspected or the scans are inconclusive.
  • Catheter venography with manometry is the reference test when stenting is being considered. A thin catheter measures the pressure on each side of the narrowing. The review says thresholds of 8–10 mm Hg are commonly used to call a gradient significant, while noting that some patients with lower gradients have also benefited.

The pressure measurement matters because a narrowing on a scan is not proof. The 2003 study found substantial narrowing in 4 of 59 people without IIH. A 2015 review in the Journal of Neuro-Ophthalmology says imaging signs such as transverse sinus stenosis are not diagnostic of IIH, and that finding them by chance should not prompt invasive procedures unless other signs, such as optic disc swelling, are present.

The size of the gradient may also predict the result. A 2019 systematic review of 186 patients found that 81 of 86 (94.2%) with a gradient above 21 mm Hg had a favourable outcome after stenting, against 82 of 100 (82.0%) with a gradient of 21 or less.

An eye examination belongs in the same workup, because swollen optic discs are what point to IIH. Whether you need a scan covers imaging criteria for tinnitus in general, and the diagnosis guide sets out what an assessment usually includes.

What venous sinus stenting is

Venous sinus stenting is an endovascular procedure: a mesh tube is guided through the blood vessels to the narrowed sinus and opened across it, holding it wide. Antiplatelet drugs, which reduce clotting, are taken for a period afterwards. A 2024 review in World Neurosurgery says there is not yet enough evidence to standardise that medication or the other decisions after the procedure.

A 2026 meta-analysis of 40 studies and 995 patients found no significant difference in outcomes between braided and laser-cut stent designs. The River stent, the first designed for the venous sinuses, was tested in a single-arm US trial of 39 people with IIH who had failed medical treatment.

What stenting achieves for the tinnitus

The best summary is the 2026 Otology & Neurotology meta-analysis, which pooled 32 studies and 850 patients: 641 with IIH and 209 with pulsatile tinnitus alone.

  • Pulsatile tinnitus improved in 89.8%.
  • It resolved completely in 87.1%.
  • Recurrence at last follow-up: 10.9%.

Results did not differ significantly between the IIH and tinnitus-only groups. Recurrence was higher in IIH, 12.0% against 5.7%, but not significantly.

Two other pooled analyses come out close to that. A 2024 analysis of 28 studies and 616 patients found improvement in 91.7%, complete resolution in 88.6% and recurrence in 6.5%. It did find a significant difference in recurrence: 10.6% in IIH against 2.0% when tinnitus was the main reason for stenting. The 2026 analysis of 995 patients reported complete resolution in 91.6%.

Figures this high are rare in tinnitus, and possible because the sound has a physical source, which puts venous sinus stenosis on the short list of causes that can be cured.

Complications, and the chance it narrows again

Serious complications are uncommon but real:

  • A 2019 meta-analysis of 474 patients with IIH found major complications in 1.9%.
  • A 2024 meta-analysis of 36 studies and 1,066 patients reported complications in 5.35%, including subdural haemorrhage (bleeding under the lining of the brain), clot forming in the stent, and urinary infection.
  • The 2026 meta-analysis of 995 pulsatile tinnitus patients put the overall complication rate at 3.6%.
  • In the River trial, the one serious adverse event was bleeding from the gut two months after the procedure, in a patient still taking two antiplatelet drugs.

The longer-term issue is renarrowing. A 2018 meta-analysis of 395 patients followed for a mean of 18.9 months found stent survival of 84%, with new narrowing next to the stent in 14%. A 2024 meta-analysis of 694 IIH patients put restenosis at 17.7%. The options then include a further stent or a shunt to drain spinal fluid, though the World Neurosurgery review says clearer criteria for retreatment are needed.

How strong the evidence is

Every outcome figure above comes from non-randomised studies of selected patients. The Frontiers review says questions remain about long-term durability, and that the role of stenting for pulsatile tinnitus without the classic features of IIH is still under investigation.

No randomised trial has reported. The protocol for the UK's IIH Intervention trial, published in 2026, states that there are no randomised data supporting any interventional approach for preserving vision in sight-threatening IIH. That trial randomises people at risk of losing vision to a spinal fluid shunt or to stenting, and its main outcome is swelling at the back of the eye, not the tinnitus.

Two more points are worth weighing. Several authors of the 2026 meta-analysis report consulting fees or equity from device companies, and the ARISE II roundtable included industry experts. That does not make the results wrong, but it is why independent trials matter. And the World Neurosurgery review notes there is no widespread agreement on who should be offered stenting: a pressure gradient and failed medical treatment are common criteria, but not every team requires the second.

For IIH itself, a 2025 review in Continuum notes that no drug is licensed, that acetazolamide is the most commonly prescribed, and that a GLP-1 drug lowered intracranial pressure in a phase II trial (GLP-1 drugs and tinnitus covers what that means for the sound). It also records rising use of stenting among patients who need surgery.

When pulsatile tinnitus needs urgent care

A narrowed venous sinus is not an emergency in itself, and pulsatile tinnitus on its own should be seen by a doctor within a few weeks.

Call your local emergency number or go to an emergency department if a pulsing sound comes with:

  • a sudden, severe headache
  • sudden neck pain
  • a drooping eyelid, loss of vision or new double vision
  • sudden weakness or drooping in the face or an arm, or trouble speaking or seeing

Not every pulsing sound comes from a vein. A tear in a neck artery, cervical artery dissection, can cause one: in a European study of 778 people with a dissection, 63 reported pulsatile tinnitus, and 41.3% of those 63 arrived with an ischaemic stroke, fewer than among the dissection patients without it (63.9%), so the study linked the pulsing sound to a milder course. A bulging artery in the brain has its own entry on intracranial aneurysm and pulsatile tinnitus, and a clot in the venous sinuses themselves, which can raise pressure inside the head and cause bleeding into the brain, is covered under cerebral venous sinus thrombosis.

If headaches, or vision that briefly greys out or blurs, have been building up alongside the whoosh, book sooner and say so. The Frontiers review lists headache, brief dimming of vision and pulsatile tinnitus among the typical symptoms of IIH, and the IIH Intervention protocol notes that its swollen optic discs put sight at risk. The when to seek care tool walks through these signs one at a time.

Questions to ask if stenting is suggested

  • Has an eye examination checked for swelling of the optic discs?
  • Was the pressure gradient across the narrowing measured, and what was it?
  • Have a fistula and a sigmoid sinus wall abnormality been looked for and ruled out?
  • Is the narrowing on the same side as the sound?
  • Which antiplatelet medicines will I take afterwards, and for how long?
  • What happens if the vein narrows again beside the stent?

Frequently asked questions

Can venous sinus stenosis cause pulsatile tinnitus?

Yes. A narrowed transverse or sigmoid sinus produces turbulent flow and higher pressure in the vein, which is heard as a whooshing in time with the pulse. A 2026 expert consensus calls venous sinus stenosis the most common cause of pulsatile tinnitus, and in a 2026 case-control study transverse sinus stenosis was significantly more common in 148 people with pulsatile tinnitus than in 98 controls. It also turns up in some people with no symptoms, so it has to be tied to the sound before anyone treats it.

How is venous sinus stenosis diagnosed?

Usually with MRI and MR venography first, which show the veins without radiation. CT venography and a fine-slice CT of the temporal bone can add detail about the bone and the degree of narrowing. If stenting is being considered, catheter venography measures the pressure on each side of the narrowing, and most centres treat a drop of about 8–10 mm Hg as significant. An eye examination for swollen optic discs is part of the same workup, because it points to raised pressure around the brain.

Does venous sinus stenting cure pulsatile tinnitus?

For most people in the published studies, yes. A 2026 meta-analysis of 32 studies and 850 patients found the tinnitus improved in 89.8% and went completely in 87.1%, with 10.9% recurrence at last follow-up. Two other pooled analyses, of 616 and 995 patients, reported similar figures. All three pool non-randomised studies of selected patients, and no randomised trial of stenting has yet reported.

What are the risks of venous sinus stenting?

Serious complications are uncommon but real. A 2019 review of 474 patients found major complications in 1.9%, and a 2024 review of 1,066 patients reported complications in 5.35%, including bleeding under the lining of the brain (subdural haemorrhage) and clot forming in the stent. Antiplatelet drugs are taken afterwards, and they carry their own bleeding risk. The vein can also narrow again beside the stent: a 2018 review found this in 14% of patients.

Is venous sinus stenosis the same as idiopathic intracranial hypertension?

No, but the two are closely linked. In a 2003 MRI study, 27 of 29 people with idiopathic intracranial hypertension had substantial narrowing of both transverse sinuses, against 4 of 59 controls. Raised pressure can squeeze the sinuses narrow, and a narrowing can in turn keep the pressure up. Stenosis also occurs without raised pressure: 209 of the 850 patients in a 2026 stenting meta-analysis had pulsatile tinnitus without intracranial hypertension.

Is pulsatile tinnitus from venous sinus stenosis an emergency?

On its own, no, but it should be seen by a doctor within a few weeks. Call your local emergency number or go to an emergency department if a pulsing sound comes with a sudden severe headache, sudden neck pain, a drooping eyelid, loss of vision or new double vision, or with sudden weakness or drooping in the face or an arm or trouble speaking. Headaches with vision that briefly greys out call for an earlier appointment.

Sources

24 named sources

Show the list
  1. Amans, Ding et al., 2026Journal article

    ARISE II Consensus on Venous Sinus Stenting, AJNR American Journal of Neuroradiology, PubMed (opens in a new tab)
  2. Alipour Khabir, Ognard et al., 2026Systematic review

    Venous Sinus Stenting for Pulsatile Tinnitus: A Systematic Review and Meta-Analysis, Otology & Neurotology, PubMed (opens in a new tab)
  3. Zhang, Klein et al., 2026Observational study

    Prevalence and Radiologic Manifestations of Internal Jugular Venous Stenosis in Patients with Pulsatile Tinnitus: A Case-Control Study, AJNR American Journal of Neuroradiology, PubMed (opens in a new tab)
  4. Terraciano, Gupta & Altschul, 2026Journal article

    Cerebral venous disorders: clinical presentation, diagnostic strategy, and contemporary management, Frontiers in Neurology, PMC (opens in a new tab)
  5. Farb, Vanek et al., 2003Observational study

    Idiopathic intracranial hypertension: the prevalence and morphology of sinovenous stenosis, Neurology, PubMed (opens in a new tab)
  6. King, Mitchell et al., 1995Journal article

    Cerebral venography and manometry in idiopathic intracranial hypertension, Neurology, PubMed (opens in a new tab)
  7. King, Mitchell et al., 2002Journal article

    Manometry combined with cervical puncture in idiopathic intracranial hypertension, Neurology, PubMed (opens in a new tab)
  8. Zhao, Jiang et al., 2021Journal article

    Why does unilateral pulsatile tinnitus occur in patients with idiopathic intracranial hypertension?, Neuroradiology, PubMed (opens in a new tab)
  9. Snowden, Van Stavern & Stunkel, 2025Journal article

    Persistence of pulsatile tinnitus in patients with idiopathic intracranial hypertension following resolution of papilledema, Journal of the Neurological Sciences, PubMed (opens in a new tab)
  10. Suresh, Srinivasan et al., 2026Journal article

    Symptomatic cerebral venous outflow resistance responds to stenting even in the absence of intracranial hypertension on manometry: a multicentre study, Neuroradiology, PubMed (opens in a new tab)
  11. Bidot, Saindane et al., 2015Journal article

    Brain Imaging in Idiopathic Intracranial Hypertension, Journal of Neuro-Ophthalmology, PubMed (opens in a new tab)
  12. McDougall, Ban et al., 2019Systematic review

    Fifty shades of gradients: does the pressure gradient in venous sinus stenting for idiopathic intracranial hypertension matter? A systematic review, Journal of Neurosurgery, PubMed (opens in a new tab)
  13. Elfadle, Hemeda et al., 2026Observational study

    Venous sinus stenting for pulsatile tinnitus secondary to venous sinus stenosis: a dual-center retrospective cohort study, Neurosurgical Review, PubMed (opens in a new tab)
  14. Schartz, Finkelstein et al., 2024Systematic review

    Outcomes of Pulsatile Tinnitus After Cerebral Venous Sinus Stenting: Systematic Review and Pooled Analysis of 616 Patients, World Neurosurgery, PubMed (opens in a new tab)
  15. Elek, Dindar et al., 2026Systematic review

    Braided Versus Laser-Cut Stents in Venous Sinus Stenting for Pulsatile Tinnitus: A Systematic Review and Meta-analysis, AJNR American Journal of Neuroradiology, PubMed (opens in a new tab)
  16. Nicholson, Brinjikji et al., 2019Systematic review

    Venous sinus stenting for idiopathic intracranial hypertension: a systematic review and meta-analysis, Journal of NeuroInterventional Surgery, PubMed (opens in a new tab)
  17. Azzam, Mortezaei et al., 2024Systematic review

    Venous sinus stenting for idiopathic intracranial hypertension: An updated Meta-analysis, Journal of the Neurological Sciences, PubMed (opens in a new tab)
  18. Saber, Lewis et al., 2018Systematic review

    Stent Survival and Stent-Adjacent Stenosis Rates following Venous Sinus Stenting for Idiopathic Intracranial Hypertension: A Systematic Review and Meta-Analysis, Interventional Neurology, PubMed (opens in a new tab)
  19. Lim, Monteiro et al., 2024Systematic review

    Stenting for Venous Sinus Stenosis in Patients With Idiopathic Intracranial Hypertension: An Updated Systematic Review and Meta-Analysis of the Literature, Neurosurgery, PubMed (opens in a new tab)
  20. Pandey, Schreiber et al., 2024Journal article

    Challenges in the use of Venous Sinus Stenting in the Treatment of Idiopathic Intracranial Hypertension and Pulsatile Tinnitus, World Neurosurgery, PubMed (opens in a new tab)
  21. Patsalides, Fargen et al., 2025Journal article

    The River study: the first prospective multicenter trial of a novel venous sinus stent for the treatment of idiopathic intracranial hypertension, Journal of NeuroInterventional Surgery, PubMed (opens in a new tab)
  22. Tsermoulas, Mollan et al., 2026Clinical trial

    Cerebrospinal fluid shunting or dural venous sinus stenting to preserve vision in idiopathic intracranial hypertension (IIH Intervention): protocol for an open-label, multicentre, randomised controlled phase IIb trial, BMJ Open, PubMed (opens in a new tab)
  23. Kellert, Kloss et al., 2016Journal article

    Prognostic significance of pulsatile tinnitus in cervical artery dissection, European Journal of Neurology, PubMed (opens in a new tab)
  24. Chen & Mollan, 2025Journal article

    Treatment and Monitoring of Idiopathic Intracranial Hypertension, Continuum, PubMed (opens in a new tab)

When to see a clinician

Call your local emergency number now if tinnitus comes with sudden weakness, numbness or drooping in the face or an arm, trouble speaking or seeing, or severe vertigo or loss of balance that will not settle. The same applies to a new pulsing sound with a sudden severe headache, sudden neck pain or a drooping eyelid. These can be signs of a stroke, or of a problem that can lead to one. The BE FAST stroke signs are in stroke and tinnitus.

Otherwise, most tinnitus is not a medical emergency. These are the patterns where a prompt assessment is worthwhile rather than something to wait out:

  • Sudden hearing loss, especially in one ear — this is treated as urgent, and the window for treatment is measured in days
  • Tinnitus that pulses in time with your heartbeat
  • Tinnitus in only one ear that persists
  • Tinnitus with episodes of dizziness or vertigo
  • Tinnitus after a head injury
  • Distress that is affecting your sleep, mood, or ability to function

To work through this properly, the when-to-see-a-doctor checklist takes each sign in turn and explains what the evidence says about it. It can raise a concern; it will never tell you that you are fine, because a checklist only knows what it asked about.

If what you need is a way to describe the impact rather than the risk, the impact self-check gives a clinician something concrete to work from. Neither tool diagnoses anything.

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